Biblio

Found 14 results
Filters: Author is Yang, Decheng  [Clear All Filters]
2014
H. M. Zhang, Dai, H., Hanson, P. J., Li, H., Guo, H., Ye, X., Hemida, M. G., Wang, L., Tong, Y., Qiu, Y., Liu, S., Wang, F., Song, F., Zhang, B., Wang, J. - G., Zhang, L. - X., and Yang, D., “Antiviral activity of an isatin derivative via induction of PERK-Nrf2-mediated suppression of cap-independent translation.”, ACS Chem Biol, vol. 9, no. 4, pp. 1015-24, 2014.
X. Ye, Zhang, H. Mary, Qiu, Y., Hanson, P. J., Hemida, M. Gomaa, Wei, W., Hoodless, P. A., Chu, F., and Yang, D., “Coxsackievirus-induced miR-21 disrupts cardiomyocyte interactions via the downregulation of intercalated disk components.”, PLoS Pathog, vol. 10, no. 4, p. e1004070, 2014.
H. M. Zhang, Qiu, Y., Ye, X., Hemida, M. G., Hanson, P., and Yang, D., “P58(IPK) inhibits coxsackievirus-induced apoptosis via the PI3K/Akt pathway requiring activation of ATF6a and subsequent upregulation of mitofusin 2.”, Cell Microbiol, vol. 16, no. 3, pp. 411-24, 2014.
2013
K. J. Jensen, Garmaroudi, F. S., Zhang, J., Lin, J., Boroomand, S., Zhang, M., Luo, Z., Yang, D., Luo, H., McManus, B. M., and Janes, K. A., “An ERK-p38 subnetwork coordinates host cell apoptosis and necrosis during coxsackievirus B3 infection.”, Cell Host Microbe, vol. 13, no. 1, pp. 67-76, 2013.
X. Ye, Hemida, M. Gomaa, Qiu, Y., Hanson, P. J., Zhang, H. Mary, and Yang, D., “MiR-126 promotes coxsackievirus replication by mediating cross-talk of ERK1/2 and Wnt/β-catenin signal pathways.”, Cell Mol Life Sci, vol. 70, no. 23, pp. 4631-44, 2013.
2012
X. Ye, Hemida, M., Zhang, H. M., Hanson, P., Ye, Q., and Yang, D., “Current advances in Phi29 pRNA biology and its application in drug delivery.”, Wiley Interdiscip Rev RNA, vol. 3, no. 4, pp. 469-81, 2012.
Z. Liu, Zhang, H. M., Yuan, J., Ye, X., Taylor, G. A., and Yang, D., “The immunity-related GTPase Irgm3 relieves endoplasmic reticulum stress response during coxsackievirus B3 infection via a PI3K/Akt dependent pathway.”, Cell Microbiol, vol. 14, no. 1, pp. 133-46, 2012.
2011
X. Ye, Liu, Z., Hemida, M. Gomaa, and Yang, D., “Targeted delivery of mutant tolerant anti-coxsackievirus artificial microRNAs using folate conjugated bacteriophage Phi29 pRNA.”, PLoS One, vol. 6, no. 6, p. e21215, 2011.
2010
H. M. Zhang, Ye, X., Su, Y., Yuan, J., Liu, Z., Stein, D. A., and Yang, D., “Coxsackievirus B3 infection activates the unfolded protein response and induces apoptosis through downregulation of p58IPK and activation of CHOP and SREBP1.”, J Virol, vol. 84, no. 17, pp. 8446-59, 2010.
2009
J. Yuan, Liu, Z., Lim, T., Zhang, H., He, J., Walker, E., Shier, C., Wang, Y., Su, Y., Sall, A., McManus, B., and Yang, D., “CXCL10 inhibits viral replication through recruitment of natural killer cells in coxsackievirus B3-induced myocarditis.”, Circ Res, vol. 104, no. 5, pp. 628-38, 2009.
2005
H. M. Zhang, Yuan, J., Cheung, P., Chau, D., Wong, B. W., McManus, B. M., and Yang, D., “Gamma interferon-inducible protein 10 induces HeLa cell apoptosis through a p53-dependent pathway initiated by suppression of human papillomavirus type 18 E6 and E7 expression.”, Mol Cell Biol, vol. 25, no. 14, pp. 6247-58, 2005.
J. Yuan, Cheung, P. K. M., Zhang, H. M., Chau, D., and Yang, D., “Inhibition of coxsackievirus B3 replication by small interfering RNAs requires perfect sequence match in the central region of the viral positive strand.”, J Virol, vol. 79, no. 4, pp. 2151-9, 2005.
2003
H. M. Zhang, Yuan, J., Cheung, P., Luo, H., Yanagawa, B., Chau, D., Stephan-Tozy, N., Wong, B. W., Zhang, J., Wilson, J. E., McManus, B. M., and Yang, D., “Overexpression of interferon-gamma-inducible GTPase inhibits coxsackievirus B3-induced apoptosis through the activation of the phosphatidylinositol 3-kinase/Akt pathway and inhibition of viral replication.”, J Biol Chem, vol. 278, no. 35, pp. 33011-9, 2003.
2002
H. M. Zhang, Yanagawa, B., Cheung, P., Luo, H., Yuan, J., Chau, D., Wang, A., Bohunek, L., Wilson, J. E., McManus, B. M., and Yang, D., “Nip21 gene expression reduces coxsackievirus B3 replication by promoting apoptotic cell death via a mitochondria-dependent pathway.”, Circ Res, vol. 90, no. 12, pp. 1251-8, 2002.